Muscle damage can be a result of injury, inactivity, or disease. Fortunately, researchers have identified a specific protein called CD163 that halts muscle repair after injury, providing some insight into possible treatments for humans with muscle damage.

Working with mice in the laboratory allowed scientists to study this protein, which is produced by white blood cells. They found that mice lacking CD163 were able to regenerate muscle much more quickly after an injury than control mice. Blood flow was also improved.

This is important, because by identifying this protein, researchers may be able to come up with ways to help human patients suffering from muscle breakdown. Read more about this research here.

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